Amyloid Beta Current Events | Amyloid Beta News | 3
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U.Va. Scientists Identify 'Missing Link' in Process Leading to Alzheimer's Disease Scientists at the University of Virginia have identified what appears to be a major missing link in the process that destroys nerve cells in Alzheimer's disease, an incurable disease that slowly destroys memory and cognitive abilities. view more (2007-02-08)
Vaccine triggers immune response, prevents Alzheimer's A vaccine created by University of Rochester Medical Center scientists prevents the development of Alzheimer's disease-like pathology in mice without causing inflammation or significant side effects. view more (2008-05-20)
Potential Alzheimer's disease drug target identified by UC San Diego researcher In findings with the potential to provide a therapy for Alzheimer's disease patients where none now exist, a researcher at the University of California, San Diego and colleagues have demonstrated in mice a way to reduce the overproduction of a peptide associated with the disease. view more (2008-03-17)
Scientists develop new treatments for Alzheimer's disease Scientists at the University of Liverpool have created a new chemical compound that could be developed into a drug treatment for Alzheimer's disease. view more (2006-10-27)
OHSU research reveals possible future target for delaying or stopping Alzheimer's Researchers at Oregon Health & Science University's Neurological Sciences Institute (NSI) have located a possible target for future therapies aimed at delaying or stopping Alzheimer's disease. view more (2006-05-02)
Jefferson Scientists Discover Mechanism Tying Obesity to Alzheimer's Disease If heart disease and diabetes aren't bad enough, now comes another reason to watch your weight. According to a study just released, packing on too many pounds can increase the risk of developing Alzheimer's disease. view more (2005-12-30)
Calorie restriction may prevent Alzheimer's through promotion of longevity program in the brain A recent study directed by Mount Sinai School of Medicine suggests that experimental dietary regimens might calm or even reverse symptoms of Alzheimer's Disease (AD). view more (2006-06-15)
Rethinking Alzheimer's disease and its treatment targets The standard explanation for what causes Alzheimer's is known as the amyloid hypothesis, which posits that the disease results from of an accumulation of the peptide amyloid beta, the toxic protein fragments that deposit in the brain and become the sticky plaques that have defined Alzheimer's for more than 100 years. view more (2009-09-23)
Alzheimer's: New findings resolve long dispute about how the disease might kill brain cells For a decade, Alzheimer's disease researchers have been entrenched in debate about one of the mechanisms believed to be responsible for brain cell death and memory loss in the illness. view more (2009-04-16)
Brain damage found in cognitively normal people with Alzheimer's marker Researchers at Washington University School of Medicine in St. Louis have linked a potential indicator of Alzheimer's disease to brain damage in humans with no signs of mental impairment. view more (2009-03-11)
Scientists demonstrate means of reducing Alzheimer's-like plaques in fly brain Neuroscientists at Cold Spring Harbor Laboratory (CSHL) are part of a collaboration that has succeeded in demonstrating that overexpression of an enzyme in the brain can reduce telltale deposits causally linked with Alzheimer's disease. view more (2008-07-16)
New findings about brain proteins suggest possible way to fight Alzheimer's The action of a small protein that is a major villain in Alzheimer's disease can be counterbalanced with another brain protein, researchers at UT Southwestern Medical Center have found in an animal study. view more (2009-10-07)
Mechanism of Alzheimer's suggests combination therapy needed Researchers at the University of Illinois at Chicago College of Medicine have discovered a mode of action for mysterious but diagnostic protein snarls found in the brains of Alzheimer's patients that suggests a one-two punch of therapy may be needed to combat the neurodegenerative disease. view more (2009-03-18)
Enzyme action creates protein linked to Alzheimer's disease Researchers at UT Southwestern Medical Center have defined a key step in the production of beta-amyloid, a short protein that is thought to be responsible for the development of Alzheimer's disease. view more (2005-08-15)
Zeroing in on Alzheimer's Hereditary Alzheimer's disease has been shown to be the result of mutations in certain specific genes. Other cases of Alzheimer's are also assumed to be traceable to the influence of a number of still unidentified genes. It is probable that these genes are located in a large area on chromosome 10q, which contains more than 100 genes. Working with... view more... (2003-09-25)
A 'grape' future for Alzheimer's disease research With National Alzheimer's Awareness Month upon us, attention continues to focus on new approaches to cognitive health in an aging population. view more (2007-11-07)
Keeping amyloid-and Alzheimer's-in check Researchers have identified a protein that reins in the rogue activity of the molecules that make the amyloid-beta protein-which may prevent normal brain function in people with Alzheimer's disease. view more (2006-04-27)
Targeting a key enzyme with gene therapy reversed course of Alzheimer's disease in mouse models In mice, that had been genetically engineered to develop Alzheimer's disease, scientists were able to reverse the rodents' memory loss by reducing the amount of an enzyme that is crucial for the development of Alzheimer's disease. view more (2005-09-22)
Alzheimer's disease therapeutic prevents long-term damage from TBI in pre-clinical studies A class of Alzheimer's disease drugs currently studied in clinical trials appears to reduce damage caused by traumatic brain injury in animals, researchers at Georgetown University Medical Center report in an upcoming advance online publication of Nature Medicine. view more (2009-03-16)
Genetic variation may reduce Alzheimer's risk Adults with a genetic variation enabling them to express higher levels of fetal hemoglobin may have a reduced risk of Alzheimer's disease, researchers say. view more (2007-01-09)
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