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Protein thought to promote cancer instead functions as a tumor suppressor, researchers report
A protein previously thought to promote colorectal cancer instead suppresses the growth of human cancer cells in culture, researchers at UT Southwestern Medical Center have found. view more (2008-07-08)
Cancer Gene Radiation Therapy Discovery
Dr Tracy Robson, a lecturer in molecular radiation science at the University's Jordanstown campus, has isolated a novel gene, called DIR-1, which can alter a tumour cell's susceptibility to radiation therapy. view more (1999-09-20)
NYU Langone Medical Center researchers identify key gene in deadly inflammatory breast cancer
Aggressive, deadly and often misdiagnosed, inflammatory breast cancer (IBC) is the most lethal form of primary breast cancer, often striking women in their prime and causing death within 18 to 24 months. view more (2009-06-15)
New Understanding Of Role Of Breast Cancer Gene In Normal Function And Disease
Researchers at the Medical Research Council (MRC) Cancer Cell Unit, Cambridge have gained an important new insight into the role of the breast cancer gene known as BRCA2. It appears to have a key function in cell division which needs to happen accurately for normal cell reproduction and repair, otherwise disease occurs. The findings are published... view more... (2004-10-28)
Translational derepression & oncogene expression in breast cancer cells
Drs. Anuradha Mehta, Christopher Trotta and Stuart Peltz (PTC Therapeutics) have uncovered a novel mechanism whereby the translation efficiency of oncogenes is increased in cancer cells. view more (2006-03-31)
Gene panel predicts lung cancer survival, study finds
Researchers from four leading cancer centers have confirmed that an analysis involving a panel of genes can be used to predict which lung cancer patients will have the worst survival. The finding could one day lead to a test that would help determine who needs more aggressive treatment. view more (2008-07-22)
Cancer cells suppress large regions of DNA by a reversible process that can be tackled
Cancer researchers at Sydney's Garvan Institute, in collaboration with Spanish scientists, have formulated a new concept for how cancer cells can escape normal growth controls, which may have far-reaching implications for the new generation of cancer therapies. view more (2006-04-24)
Large study documents how p53 mutations link to high-grade breast cancer, poor outcomes
In what is believed to be the largest study of its kind in the US, researchers have found that almost 26 percent of women studied who have breast cancer have mutations in a gene important in controlling cell growth and death, and that patients with mutations in this gene -- known as p53 - had poorer outcomes including a significantly increased... view more... (2009-04-20)
Breast cancer gene increases risk of several cancers in men
A genetic mutation implicated in an increased risk of breast and ovarian cancers also significantly increases the risk of pancreatic and prostate cancers in men, finds research in the Journal of Medical Genetics. view more (2005-09-02)
A mutation that causes resistence to chemotherapy treatment of lung cancer has been discovered
Lung cancer, mainly caused by tobacco and inhaling radon gas, is the most lethal cancer in the western world. In Spain alone this disease causes around 15,000 deaths each year. view more (1999-09-27)
Blocking one protein helps cancer cells die of natural causes
Researchers have identified a protein fragment that keeps at least one major tumor suppressor gene from preventing cancer like it should. The fragment belongs to a class of proteins known as apoptotic enhancers (ASPP), named for their ability to stimulate programmed cell death, or apoptosis, by the p53 gene. But a study published in Nature... view more... (2003-01-08)
Researchers track down the genes that could put the brakes on breast cancer.
Normally, old or damaged cells are told to stop dividing and 'self-destruct' to prevent any mutations being duplicated and growing into tumours. But cancer cells usually ignore these messages and become 'immortal' allowing them to continue multiplying indefinitely and out of control. Scientists already know that an enzyme called 'telomerase' is... view more... (1999-02-22)
Researchers identify new drug targets for cancer
Solving a 100-year-old genetic puzzle, researchers at the University of California, San Diego (UCSD) School of Medicine have determined that the same genetic mechanism that drives tumor growth can also act as a tumor suppressor. view more (2007-01-02)
Linchpin gene may be useful target for new breast cancer therapies
University of Iowa researchers have discovered a gene that plays a linchpin role in the ability of breast cancer cells to respond to estrogen. The finding may lead to improved therapies for hormone-responsive breast cancers and may explain differences in the effectiveness of current treatments. view more (2007-09-17)
Markers that can predict cancer spread could save women from unnecessary chemotherapy
Women with early breast cancer could avoid needless chemotherapy thanks to work carried out in Chicago on identifying biochemical markers which indicate whether or not cancer is likely to spread to other parts of the body, the 3rd European Breast Cancer Conference in Barcelona heard today (Saturday 23 March). Ruth Heimann, Associate Professor in... view more... (2002-03-21)
Researchers identify another potential biomarker
Researchers from Boston University School of Medicine (BUSM) have demonstrated that a recently discovered class of molecule called microRNA (miRNAs), regulate the gene expression changes in airway cells that occur with smoking and lung cancer. view more (2009-01-14)
Reversing effects of altered enzyme may fight brain tumor growth
An international team of scientists from the Moores Cancer Center at the University of California, San Diego, the University of North Carolina and several institutions in China have explained how a gene alteration can lead to the development of a type of brain cancer, and they have identified a compound that could staunch the cancer's growth. view more (2009-04-14)
Study finds BRAF mutations in colorectal cancer cause resistance to anti-EGFR therapy
European researchers have found that metastatic colorectal cancer patients with a mutation in the BRAF gene do not respond to anti-EGFR therapy with cetuximab and panitumumab. view more (2008-10-23)
Standard treatment for prostate cancer may encourage spread of disease
A popular prostate cancer treatment called androgen deprivation therapy may encourage prostate cancer cells to produce a protein that makes them more likely to spread throughout the body, a new study by Johns Hopkins researchers suggests. view more (2007-10-01)
Second gene linked to familial testicular cancer
Specific variations or mutations in a particular can gene raise a man's risk of familial, or inherited, testicular germ-cell cancer, the most common form of this disease. view more (2009-06-30)
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