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A genome wide search for genes underlying anxiety disorders turned up unexpected candidates
Increasing the activity of two enzymes better known for their role in oxidative stress metabolism turns normally relaxed mice into "Nervous Nellies," according to research conducted at the Salk Institute for Biological Studies and reported in the early online edition of Nature.   view more (2005-10-31)

Transporter is possible target for safer pain medicine
A transporter that silences one of the body's natural pain killers holds promise for new powerful, non-addictive pain medicines as well as understanding AIDS patients' increased pain perception.   view more (2006-06-12)

Mouse model of Parkinson's reproduces nonmotor symptoms
The classic symptoms of Parkinson's disease involve tremor, stiffness and slow movements. Over the last decade, neurologists have been paying greater attention to non-motor symptoms, such as digestive and sleep problems, loss of sense of smell and depression.   view more (2009-06-23)

Study suggests new treatments for Huntington's disease
Working with fruit flies, researchers have discovered a new mechanism by which the abnormal protein in Huntington's disease causes neurodegeneration.   view more (2008-01-10)

Deafness and seizures result when mysterious protein deleted in mice
Scientists have discovered that mice genetically engineered to lack a particular protein in the brain have profound deafness and seizures. The finding suggests a pathway, they say, for exploring the hereditary causes of deafness and epilepsy in humans.   view more (2008-01-25)

New protein synthesis not essential to memory formation
New research from the University of Illinois challenges the premise that the brain must build new proteins in response to an experience for that experience to be recorded in long-term memory.    view more (2007-07-27)

Einstein scientists discover cause and possible treatments for hereditary movement disorder
Researchers at the Albert Einstein College of Medicine of Yeshiva University have discovered the underlying cause of a type of ataxia, hereditary disorders characterized by poor balance, loss of posture and difficulty performing rapid coordinated movement.   view more (2006-03-02)

Stress triggers relapse in meth abuse, OHSU study finds
Oregon Health & Science University research showing stress triggers a relapse of methamphetamine abuse in mice could be a step toward developing a drug to curb this frustrating obstacle to recovery.   view more (2006-10-19)

Diabetes Slows Nerve Recovery After Heart Transplant
Diabetes has a detrimental effect on a person's ability to recover from a heart transplant, notes a study in the September Journal of Nuclear Medicine.   view more (2006-09-06)

Do 'light' cigarettes deliver less nicotine to the brain than regular cigarettes?
For decades now, cigarette makers have marketed so-called light cigarettes - which contain less nicotine than regular smokes - with the implication that they are less harmful to smokers' health. A new UCLA study shows, however, that they deliver nearly as much nicotine to the brain.   view more (2008-09-29)

Botulism study could lead to new vaccines and treatments to counter bioterrorist attacks
Of all the weapons in the bioterrorist arsenal, none is as potent as botulinum neurotoxin, which causes botulism-a potentially fatal disease with symptoms that include severe paralysis of the limbs and respiratory muscles.   view more (2006-12-14)

Mice provide important clues to obsessive-compulsive disorder
Mice born without a key brain protein compulsively groom their faces until they bleed and are afraid to venture out of the corner of their cages.   view more (2007-08-23)

Brain works more chaotically than previously thought
The brain appears to process information more chaotically than has long been assumed. This is demonstrated by a new study conducted by scientists at the University of Bonn.   view more (2007-02-28)

New Target Found to Fight, Treat Parkinson's
Neuroscientists from the University at Buffalo have described for the first time how rotenone, an environmental toxin linked specifically to Parkinson's disease, selectively destroys the neurons that produce dopamine, the neurotransmitter critical to body movement and muscle control.   view more (2005-08-25)

Researcher finds new use for botox
Botox, used by Hollywood stars to smooth out facial wrinkles, is playing an important role in UQ research to understand how nerve cells communicate with each other.   view more (2005-09-22)

Researchers separate analgesic effects from addictive aspects of pain-killing drugs
For the first time, pain researchers at Washington University School of Medicine in St. Louis have shown that it's possible to separate the good effects of opiate drugs such as morphine (pain relief) from the unwanted side effects of those drugs (tolerance, abuse and addiction).   view more (2007-08-22)

Nicotine Lessens Symptoms of Depression in Nonsmokers
Nicotine may improve the symptoms of depression in people who do not smoke, Duke University Medical Center scientists have discovered.   view more (2006-09-13)

Scientists reveal how deadly toxin hijacks cells
Scientists have pinpointed exactly how botulinum neurotoxin A-a potential agent of biological warfare and one of the most lethal toxins known to man-is able to sneak into cells.   view more (2006-03-17)

The secret of internal bliss revealed
Scientists now know for sure how cannabis works in the brain. By studying naturally occuring cannabinoids they have found that cannabis 'speaks' to nerve cells instructing them to stop releasing their chemical neurotransmitters so dumbing down their effects. The upside is that it makes cannabis effective for treatment of MS but it also has a... view more... (2001-04-03)

Dying of excitement
For neurons, overexcitement is deadly. To avoid this, brain cells must sop up unneeded neurotransmitters from the synapse through membrane-bound transporters. If these transporters fail, neurons and other brain cells get excited to death- a phenomenon that may contribute to brain damage during stroke and Alzheimer's disease.   view more (2006-03-06)
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