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Current Chemokines News and Events, Chemokines News Articles.
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Jefferson scientists find how HIV causes dementia
Ever since the AIDS epidemic began more than two decades ago, scientists have been trying to understand why as many as one-quarter of those infected with HIV develop dementia. Now, researchers have shown that the virus produces proteins that turn on specific biochemical pathways in the brain, leading to brain cell death. The findings could lead to improved drugs to protect brain cells. (2004-04-19)

A viral cure for type 1 diabetes
Viruses can both cause and prevent autoimmune disease. In order to understand this dualism, Matthias von Herrath and colleagues from the La Jolla Institute for Allergy and Immunology in California exposed prediabetic mice to viral infections. In the January 2 issue of the Journal of Clinical Investigation the authors report that infection with lymphocytic choriomeningitis virus (LCMV) during the prediabetic period completely abolished the diabetic process in two distinct mouse models. (2004-01-02)

U.Va. researchers uncover role of platelets in hardening of the arteries
Scientists at the University of Virginia School of Medicine have discovered a new contributor to atherosclerosis, the most common form of hardening of the arteries. Doctors at U.Va. say research on mice has determined for the first time that activated platelets circulating in the blood, long understood as markers for atherosclerosis, really serve as participants in the process that eventually leads to atherosclerosis. (2002-12-15)

New hope for vaccine to fight rheumatoid arthritis, other autoimmune diseases
Promising animal test results that could lead to a vaccine to fight rheumatoid arthritis, multiple sclerosis (MS) and other autoimmune diseases were announced by researchers at the Technion-Israel Institute of Technology. The vaccine reversed an arthritic-like disease in rats by mobilizing part of the immune system to protect joints under attack by other immune cells. (2002-10-28)

Molecular mechanism underlaying anthrax infection described by UCSD School Of Medicine researchers
The mechanism by which inhaled anthrax disarms and evades the immune system, enabling the potentially lethal bacteria to rapidly spread throughout the body, has been described by researchers at UCSD School of Medicine. (2002-08-29)

Chemical attraction needed to launch an immune attack
A team led by UCSF scientists has determined how the weapons producers of the immune system - the B cells that make antibodies - find the T cells they must team up with to attack invading pathogens. The discovery may provide a strategy to block autoimmune diseases, the study's leaders suggests. (2002-03-06)

New compounds suppress neuroinflammation of Alzheimer's disease
Researchers from Northwestern Univesity and the Universite' Louis Pasteur have synthesized a new class of chemicals which suppress the cellular signaling processes that trigger inflammation of brain cells, a hallmark of Alzheimer's disease, stroke and other neurodegenerative diseases. (2002-01-23)

UC Riverside scientists discover wound-healing substance
New research with chickens at the University of California, Riverside has identified a protein pivotal in healing the animals' injuries. The discovery may help researchers understand how wound-healing can be accelerated in humans. (2002-01-23)

UT Southwestern researchers develop new method of delivering vaccines
Researchers at UT Southwestern Medical Center at Dallas have developed a quicker, more cost-efficient method of delivering dendritic cell-based vaccines - a discovery that moves anti-tumor vaccines closer to a practical reality. (2001-12-28)

The immune system and Alzheimer's disease
Utrecht researchers, funded by NWO, have determined the role played by brain cells from the immune system that are located close to dying memory cells. The research will help determine the causes of Alzheimer's disease. (2001-11-26)

Excess iron intake increases risk of intestinal infections, study suggests
Researchers here believe that an overdose of iron in the nation's diet could be rendering thousands of otherwise healthy people prone to intestinal infection. In a laboratory study, the researchers found that human intestinal cells with excess iron were more susceptible to attack by bacteria that cause infection of the small intestine. (2001-10-15)

Duke researchers shed light on a depression in cells that serves as a route of entry for pathogens and toxins
Microbiologists at Duke University Medical Center show from a review of studies that a puzzling depression in cells may serve as an invading pathway for pathogens such as the AIDS virus HIV and other viruses and toxins. (2001-08-23)

Chemokines in allergic asthma
The recognition of two major classes of helper T cells, Th1 and Th2, has focused attention on the distinctive sets of cytokines produced by these cells and on their role in activating or suppressing inflammatory responses. More recently, it has become clear that Th1 and Th2 cells also differ with respect to the chemokine receptors they express, a finding that may help explain the different timing of their entry into inflamed tissues. (2001-05-29)

Study identifies culprits in food allergy inflammation
A new study has definitively linked a type of food allergy whose occurrences have risen dramatically in recent years to eosinophils, a type of cell usually found in the blood. The study lays the foundation for the development of new drugs that block this critical food allergy pathway. (2001-03-27)

Chemokines and angiostasis
Romagnani et al. report that human microvascular endothelial cells from a variety of normal tissues express the chemokine receptor CXCR3, several of whose ligands are strongly angiostatic. This pathway could provide an additional mechanism to block tumor angiogenesis. (2000-12-31)

Scientists discover new molecule of the immune system: A chemokine that binds one of the HIV co-receptors
A team of scientists at the University of California, San Francisco and Compugen, Ltd. has discovered a new molecule of the immune system -- a member of a family of proteins called chemokines which recruit the body's army of defensive immune cells to sites of invasion. (2000-10-04)

Naked vaccination may conquer arthritis and MS
A modification of a new technology born of genetic engineering -- known as naked DNA vaccination -- holds the potential of overcoming autoimmune diseases such as multiple sclerosis and rheumatoid arthritis. It consists of introducing engineered genes based on the culprit peptides into the body. When the patient's immune system sees these genes as outside invaders, it produces its own neutralizing antibodies capable of restraining the disease. (2000-08-01)

Cincinnati Children's researchers uncover critical food allergy pathway
Researchers have identified a critical pathway that plays a key role in the development of food allergy. The discovery could lead rapidly to clinical trials of new drugs that would block the protein eotaxin, thereby preventing allergic reactions in the gastrointestinal tract (2000-06-05)

MGH study shows protein can repel as well as attract immune cells
Researchers from the Massachusetts General Hospital are the first to show that a chemical signal controlling the movement of immune system cells can repel those cells as well as attracting them. The study opens up the possibility of new ways of manipulating the immune system. (2000-05-01)

Duke researchers discover potent ability of specific immune system cell against HIV
Scientists have long known that immune system cells known as (2000-03-26)

'Playing in the sandbox' yields major AIDS breakthrough
Rajeet Pannu at the University of Alberta, along with Dr. Alshad Lalani find that a relative of human smallpox specific to rabbits, called myxoma, infects cells through the same entryway used by the HIV. If scientists can shut the door on myxoma, it may do the same for AIDS and possibly create, down the road, an AIDS vaccine. (1999-12-20)

Oregon Health Sciences University researchers find a genetic link between vascular disease and a common human herpes virus
OHSU researchers have found a connection between human cytomegalovirus and vascular disease. The cytomegalovirus- linked vascular disease surfaces in patients who have undergone an organ transplant or angioplasty procedure.Cytomegalovirus has already infected up to 50 percent of the adult population in the United States. (1999-11-23)

Fauci To Present New Insights Into HIV Latency At 12th World AIDS Conference
New data show that latent pools of infected cells are established very early in the course of HIV infection, even if a patient is treated expeditiously with highly active antiretroviral therapy. Anthony S. Fauci, M.D., NIAID director, will present these and other new findings at the 12th World AIDS Conference in Geneva, Switzerland. (1998-06-30)

Green Light For Chemokine Receptors
Potent inhibition of HIV-1 entry into macrophages has been demonstrated for the first time. A Glaxo Wellcome collaborative study showed that a chemically modified chemokine, AOP-RANTES, inhibited HIV-1 entry not only into lymphocytes but also into macrophages. This inhibition was achieved at the nanomolar level (1997-04-09)

Serendipity Bridges Gap Between Research On HIV Receptors And Antibodies
In 1996, a group of cell-surface receptors was discovered that, along with the receptor CD4, must be present for HIV infection. During the same period, James A. Hoxie, MD, an associate professor of medicine, developed an antibody against one of these important receptors -- although this fact was not immediately apparent. (1997-04-01)

Onslaught Of Full-Blown AIDS May Signal New Immune-Cell Targets: Chemokine Receptors Provide Point Of Entry For Different Virus Invaders
Harvard Medical School researchers have made a discovery helping to explain why the immune system of AIDS patients collapses after years of infection. They report in the March 4 Proceedings of the National Academy of Sciences that strains of virus developing later in patients may attack a different, larger population of immune cells through different chemokine receptors. (1997-03-04)

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