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Novel structural insights into Phytophthora effectors challenge long-held assumptions in plant pathology

Researchers at FABI define a conserved subset of Phytophthora RxLR effectors with short linear motifs embedded within folded WY domain cores. This arrangement preserves domain integrity while enabling potential interactions with host immune components, reframing pathogen strategies and challenging SLiM dogma.

SourceAmerican Phytopathological Society·JournalMolecular Plant-Microbe Interactions·DateFeb 24, 2026

Blast from the past

Researchers have discovered a new source of resistance to the devastating wheat blast disease, leveraging a gene that also protects against powdery mildew. The Pm4 gene, found in European wheat varieties, confers dual protection against the pathogen and its effector molecule AVR-Rmg8.

SourceJohn Innes Centre·JournalNature Plants·TypeExperimental study·DateJun 20, 2024
Apple MacBook Pro 14-inch (M4 Pro)

Apple MacBook Pro 14-inch (M4 Pro) powers local ML workloads, large datasets, and multi-display analysis for field and lab teams.

Sweetpotato’s sweet revenge

Researchers have identified 31 effector genes from the fungus Ceratocystis fimbriata, which causes devastating black rot in sweetpotatoes. This breakthrough provides a new approach to developing disease-resistant crops using effector-assisted breeding.

SourceAmerican Phytopathological Society·JournalMolecular Plant-Microbe Interactions·DateJun 12, 2024

Toronto researchers devise new way to find proteins for targeted treatment of disease

Researchers at the University of Toronto and Sinai Health have created a new platform to identify proteins that can be co-opted to control the stability of other proteins. The study identified over 600 new effector proteins that could be used therapeutically, including those that can efficiently degrade or stabilize target proteins.

SourceUniversity of Toronto·JournalNature·TypeExperimental study·DateMar 22, 2024
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Study IDs secret of stealthy invader essential to ruinous rice disease

Researchers have identified an essential stage in the takeover of rice cells by a fungus, which could accelerate treatment or prevention of rice blast disease. The discovery involves a modification in tRNA molecules that aid in protein construction, and its absence leads to reduced virulence.

SourceUniversity of Nebraska-Lincoln·JournalNature Microbiology·DateAug 24, 2023

Prestigious support for new concepts in RNA research

Researchers investigate how bacteria modify host RNA using effector proteins to ensure their survival, a process previously unknown in eukaryotes. The team aims to decipher the mechanisms behind this process and its benefits for the bacteria.

SourceJohannes Gutenberg Universitaet Mainz·DateJul 27, 2023

A single molecule upsets symbiosis

A recent study has shown that the mutual symbiosis between bacteria and fungi can be fragile, as a specific protein maintains the balance. When this protein is absent, the bacteria are trapped within fungal hyphae and die.

SourceLeibniz Institute for Natural Product Research and Infection Biology - Hans Knöll Institute -·JournalCurrent Biology·TypeExperimental study·DateJul 5, 2023
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Fungus has a host of issues

Researchers identified four fungal proteins responsible for suppressing host plant immunity in infectious diseases, leading to distinct host specificity in over 70% of plant diseases. Understanding the mechanism of this specificity may lead to new crop protection technologies.

SourceKyoto University·JournalNew Phytologist·TypeExperimental study·DateJun 1, 2023

The fungal effector Rip 1 suppresses maize host defense responses

The Ustilago maydis effector Rip1 targets and binds Zmlox3, a maize gene from the lipoxygenase family, to suppress PTI and reduce susceptibility to fungal infection. This action leads to reduced ROS-burst formation in infected plant cells, highlighting the complex co-evolutionary forces between host and pathogen.

SourceLeibniz Institute of Plant Genetics and Crop Plant Research·JournalThe Plant Cell·DateMay 5, 2022

Infectious bacteria force host plants to feed them, study finds

Researchers discovered that bacterial virulence factor WtsE initiates mobilization of nutrients and water into spaces where the bacteria reside in infected maize plants. This process precedes death of plant cells and could inform future breeding practices to resist devastating corn diseases.

SourceOhio State University·JournalCell Host & Microbe·DateApr 13, 2022
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SAMSUNG T9 Portable SSD 2TB transfers large imagery and model outputs quickly between field laptops, lab workstations, and secure archives.

Bacteria’s hidden weapon: Toxins locked inside a capsule secured by a cork

Researchers have gained a deeper understanding of how bacteria use the type VI secretion system to develop toxins for battle. The discovery reveals that toxins are encapsulated in a capsule secured by a cork-like plug, which can be released upon mechanical force.

SourceMax Planck Institute of Molecular Physiology·JournalPLOS Pathogens·TypeExperimental study·DateFeb 9, 2022

Battle of the Pleiades against plant immunity

A group of corn smut proteins, known as the Pleiades, launch a battle against maize immunity by targeting key defense mechanisms. The study reveals that eight of the ten Pleiades inhibit reactive oxygen species production, while two others promote flowering by dampening immunity.

SourceGregor Mendel Institute of Molecular Plant Biology·JournalPLOS Pathogens·DateJun 24, 2021

Essential virulence proteins of corn smut discovered

A complex of seven proteins is essential for Ustilago maydis to infect its host plant maize. The discovery could lead to developing new fungicides and understanding how effectors function in biotrophic fungi.

SourceMax-Planck-Gesellschaft·JournalNature Microbiology·DateMay 7, 2021

Fantastic voyage: Nanobodies could help CRISPR turn genes on and off

Stanford researchers developed a novel technique attaching nanobodies to CRISPR for targeted gene control. This combo enables precise on/off switching of specific genes, potentially correcting epigenetic defects without combining large effectors.

SourceStanford University School of Engineering·JournalNature Communications·DateFeb 24, 2021
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Phytoplasma effector proteins devastate host plants through molecular mimicry

A team of biologists discovered that phytoplasma effector proteins interact with specific molecules in plant hosts, causing developmental abnormalities and devastating changes. The research found that the effector proteins adopt a structure similar to their target host molecules, allowing them to bind and cause harm.

SourceAmerican Phytopathological Society·JournalMolecular Plant-Microbe Interactions·DateOct 26, 2020

Plants can skip the middlemen to directly recognize disease-causing fungi

Researchers found that multiple variants of the same resistance gene can bind dissimilar pathogen proteins in distantly related plant species, enabling direct recognition of disease-causing fungi. This discovery has significant implications for generating disease-resistant crops and could lead to rationally designed synthetic receptors.

SourceMax Planck Institute for Plant Breeding Research·DateFeb 19, 2019

Molecular virologist fights influenza at the molecular level

Researchers have identified two small-molecule experimental inhibitors that target the influenza protein NS1, which plays a crucial role in blocking the body's immune response. The study's findings provide strong evidence for the mechanism of action of these compounds and offer significant structural insights into NS1.

SourceUniversity of Alabama at Birmingham·JournalJournal of Biological Chemistry·DateNov 2, 2018
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When foes become friends

Researchers discovered that a few changes in the genome are sufficient to turn a fungal plant pathogen into a potentially beneficial organism. The beneficial fungus has gained new genes and lost others, leading to reduced effector proteins needed to suppress the plant's immune system.

SourceMax-Planck-Gesellschaft·JournalNature Communications·DateMay 12, 2016

Uncoding a citrus tree killer

A team of researchers led by UC Riverside scientist Wenbo Ma has received a $4 million grant to study the citrus greening disease and develop resistant varieties. They will use CRISPR-based genome editing to modify native citrus genes and investigate public acceptance of genome-edited crops.

SourceUniversity of California - Riverside·DateFeb 9, 2016

Faster, not stronger: How a protein regulates gene expression

Researchers at EPFL have discovered how a major effector protein regulates gene expression by speeding up its search for chromatin binding sites. By increasing its binding rate and forming dimers to maximize interaction with chromatin, HP1α enhances gene regulation efficiency.

SourceEcole Polytechnique Fédérale de Lausanne·JournalNature Communications·DateJun 18, 2015
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Scientists identify genetic mechanism that contributed to Irish Famine

A team of researchers at the University of California, Riverside, has discovered a genetic mechanism that explains how Phytophthora pathogens compromised the potato plant's immune system during the Irish Famine. The study reveals that RNA silencing pathways are suppressed by effectors, leading to an increase in susceptibility to disease.

SourceUniversity of California - Riverside·JournalNature Genetics·DateFeb 6, 2013

ISU plant pathologist updates science community on groundbreaking research

Researchers have built upon the 2009 discovery of TAL effector proteins, which enable targeted gene manipulation, leading to breakthroughs in understanding gene function and improving traits in livestock and plants. The technology has also been successfully used in model organisms such as yeast, zebrafish, and human stem cells.

SourceIowa State University·JournalScience·DateOct 11, 2011
Apple iPad Pro 11-inch (M4)

Apple iPad Pro 11-inch (M4) runs demanding GIS, imaging, and annotation workflows on the go for surveys, briefings, and lab notebooks.

How plague-causing bacteria disarm host defense

Yersinia pathogen uses effector protein YpkA to target Gaq, a messenger protein that transmits alarm signals into the host cell. This study identifies a novel molecular target for preventing disease and fighting antibiotic-resistant strains.

SourceUniversity of California - San Diego·JournalMolecular Cell·DateMay 24, 2007