Articles tagged with Central Amygdala
Researchers will examine whether online support strengthens brain development linked to emotional and mental health in prematurely born infants. The scalable intervention could transform how families receive support after discharge from the neonatal intensive care unit, bringing evidence-based support into the home.
Researchers developed piezoelectric patch sensors that track finger movements to play rock-paper-scissors, promoting self-motivated healthcare and entertainment. The sensors provide quantifiable data and objective feedback, improving patient outcomes through accessibility, independence, and motivation.
Researchers at the University of Houston found that distraction disrupts memory consolidation primarily due to demands on central executive processing. To improve short-term memory, focus attention on a task for a few seconds before switching, and avoid multitasking.
A new study identifies a previously unknown brainstem pathway controlling hand and arm movements, revealing a multi-stage pathway integrating signals from the cortex, brainstem, and spinal networks. This finding may lead to new therapies for stroke rehabilitation, providing additional targets for neuromodulation treatments.
A study published in Science Advances reveals that an autism-linked mutation disrupts brain circuits responsible for erasing fear memories, leading to PTSD-like symptoms. By reactivating specific neurons, researchers were able to reverse the behavioral and physiological abnormalities.
Researchers discovered that activating NPBWR1 neurons in the central nucleus of the amygdala enhances social interactions with novel individuals. This study also found that overexpressing the human NPBWR1 gene in mouse neurons reduces sociability, highlighting the importance of genetic variations in personality traits.
Researchers have discovered that the amygdala uses specialized neurons to learn about threats and rewards, a finding that could lead to more effective treatments for anxiety and drug addiction. By studying how these neurons respond differently to various stimuli, scientists have gained insights into the brain's reward system.
Researchers found that suspected amygdala neurons producing corticotropin-releasing factor are not necessary for alcohol withdrawal or dependence. Instead, the CRF is believed to come from other neurons in the brain, with potential implications for human treatment options.
A new study by the University of Arizona has identified a brain region and neural circuitry that mediate satiation, making it possible to create treatments with fewer side effects for eating disorders and weight management. The parasubthalamic nucleus plays a key role in regulating feelings of fullness after eating.
A study published in Science Advances reveals a previously unknown mechanism behind compulsive alcohol use, which may be targeted by medication. A small group of nerve cells in the central amygdala promote alcohol use despite negative consequences.
A recent study found that neuronal microcircuits in the amygdala play a crucial role in regulating fear responses and suppressing fear memories. The researchers' findings suggest that dysfunction of this system can contribute to anxiety disorders, paving the way for more effective therapies.
New research reveals a strong correlation between opioid use, brain inflammation, and negative emotional states during withdrawal. The study found that inflammation in the gut and central amygdala may contribute to the severity of withdrawal symptoms.
Researchers at Cold Spring Harbor Laboratory have identified a neural circuit in the amygdala that generates anxiety. The critical role of dynorphin was revealed, which could serve as a cellular target for treating anxiety-related disorders.
A new study maps distinct neighborhoods where feelings are assigned to experience in the basolateral amygdala, revealing a region with diverse and dynamic neurons that interact with each other. The findings shed light on how valence assignment works and may provide insights into mental health disorders.
Research in mice reveals that the central amygdala, not the lateral portion, is crucial for aversive learning and associating sensory stimuli with threat. This finding has implications for treating disorders like anxiety and PTSD by modifying fear memories.
Researchers found a circuit in the central amygdala that responds to rewarding events and stimulates reward-related behavior, contradicting the consensus that the structure is primarily linked with fear.
A team of researchers at Cold Spring Harbor Laboratory has identified a new pathway that controls fear memories and behavior in the mouse brain. The discovery, published in Nature, reveals that the paraventricular nucleus of the thalamus plays a critical role in regulating fear learning and memory.
Researchers identified the neurofibromatosis type 1 (Nf1) gene as a key regulator of gamma-aminobutyric acid (GABA), a neurotransmitter that lowers anxiety and increases feelings of relaxation. Variations in the human Nf1 gene are linked to alcohol-dependence risk and severity, suggesting a potential genetic basis for excessive drinking.
Researchers at Cold Spring Harbor Laboratory have identified a novel neural circuit linking the amygdala with the brainstem's fear response center. This discovery sheds light on how fear is translated into behavioral responses, paving the way for potential treatments of anxiety disorders like PTSD.
Researchers have confirmed the importance of the nociceptin system in modulating the effects of stress. The study found that this system can prevent and even reverse some cellular effects of acute stress in an animal model.
A recent study by Cold Spring Harbor Laboratory researchers has identified a specific class of neurons in the central amygdala as crucial for encoding and recalling fear memories. The team used optogenetics to activate these neurons, which release neurotransmitters that enhance or inhibit synaptic connections, leading to fear responses.
A team of Scripps Research scientists found a key biological mechanism underpinning the transition to alcohol dependence. They discovered that neuropeptide Y, a naturally occurring anti-stress agent, plays a crucial role in mediating the transition to alcohol dependence by modulating GABA release in the amygdala.
A Scripps Research study has found that the endocannabinoid system, which regulates memory and emotional reactions, can also dampen the effects of alcohol. The research suggests a potential target for developing drugs to treat alcohol addiction.