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Friedrich Miescher Institute


A new mechanism for accessing damaged DNA

Researchers from the Thomä group at FMI have identified a new mechanism by which UV-DDB detects and binds to damaged DNA tightly packed in nucleosomes. This mechanism, known as 'slide-assisted site-exposure', allows repair proteins to bind to lesions without requiring additional proteins or chemical energy.

SourceFriedrich Miescher Institute·JournalNature·DateMay 30, 2019

Stressed, anxious? Ask the brain!

Researchers used deep brain imaging to study amygdala activity in mice, revealing two large antagonistic sets of neurons that encode opposing moment-to-moment state changes. The findings suggest a complex neural basis for internal states like anxiety, which may have implications for understanding human anxiety disorders.

SourceFriedrich Miescher Institute·JournalScience·DateApr 23, 2019

Clues for drugging the 'undruggable'

Researchers have identified a new way to target and degrade a class of proteins called zinc finger transcription factors, which play critical roles in health and disease. By modifying thalidomide analogs, scientists can selectively degrade specific zinc fingers, offering a promising lead for developing new cancer treatments.

SourceFriedrich Miescher Institute·JournalScience·DateNov 5, 2018

Circuitry of fear identified

Neurobiologists have identified specific types of neurons in the amygdala that process fear inputs and regulate subsequent fear responses. These circuits play a key role in the generalization of fear, allowing it to become divorced from its original situation, and may provide new targets for treating phobias and anxiety disorders.

SourceFriedrich Miescher Institute·JournalNature·DateNov 11, 2010