Articles tagged with Cellular Neuroscience
Researchers have created 3D images of fruit fly brains using optical projection tomography, shedding light on genetic research into Alzheimer's and other human diseases. The images allow scientists to visualize gene expression patterns and gain insights into the human brain.
Dr. John H. Morrison has received a $5 million MERIT Award to support his research on brain cell adaptability and its impact on age-related cognitive decline. This recognition highlights the strength of Mount Sinai faculty during a challenging time for researchers.
Researchers have found that proteins essential for brain growth during development are equally important for forming long-term memories. Growth factors like BDNF play a critical role in retaining long-term memory by binding to specific receptors in the brain, and their absence can block this process.
Frontotemporal dementia is caused by defects in chromosome 17, specifically in the progranulin protein. Genetic analyses identified a shortage of progranulin in FTDU families, leading to brain cell death. This discovery offers new diagnostic and therapeutic possibilities for FTD and other brain diseases.
A new study has found a significant link between lead exposure and long-term brain cell loss and damage. Researchers measured the brains of former employees exposed to lead in their workplaces, finding those with higher levels had smaller brain volumes and more damage.
Researchers found that people taking all three medications experienced less severe strokes, shorter hospital stays, and better brain function after a stroke. The study suggests that these medications may work together to improve blood flow and reduce brain cell damage.
Researchers accurately predicted fMRI signals in patients while watching a movie, validating its use in human neuroscience research. The findings also highlight the importance of further study to understand the correlation between fMRI and single neuronal activity.
Researchers found that clearing Alzheimer's plaques leads to rapid recovery of normal brain structure in just three days. The study suggests that plaque-clearing treatments may slow or halt disease progression, and researchers plan to investigate the underlying mechanisms.
Researchers at Johns Hopkins Medicine have made a groundbreaking discovery that prostaglandin-E2 (PGE2) protects brain cells from damage caused by stroke. The finding provides a new strategy for tackling and understanding the condition, which affects 4 million Americans annually.
Scientists at University of Toronto have found a major mechanism causing brain cells to die after stroke, leading to the development of new treatments. They discovered that the TRPM7 channel activates a lethal chain reaction when oxygen and nutrients are deprived, resulting in massive free radical production and cell death.
Researchers at Johns Hopkins have identified a novel gene mutation causing Huntington's Disease-like 2 (HDL2), a condition identical to Huntington's but caused by a different mutation. The discovery provides a window into the mechanisms of brain cell death and could shed light on other neurodegenerative disorders.
A study published in Radiology found that Gulf War veterans experienced significant brain-cell loss, particularly in the brain stem, right basal ganglia, and left basal ganglia. The loss of functioning brain cells was linked to various symptoms, including joint pain, fatigue, dizziness, and mental confusion.
Researchers found changes in brain cells in rats treated with anti-depressant and obesity drugs, including shriveling and abnormal corkscrew shapes. The study raises concerns about the prolonged use of such commonly prescribed drugs as fluoxetine and sertraline.
Lab tests conducted by Allan Butterfield show that vitamin E can prevent the death of brain cells exposed to a toxic protein found in Alzheimer's brains. The study suggests that antioxidants may be a key to unlocking better therapies for the devastating disease.