Articles tagged with Long Term Potentiation
Researchers discovered astrocytes actively preserve long-term memories by regulating Ank2 and BDNF signaling. This finding expands the understanding of how memories are stored in the brain.
A new study reveals that astrocytes actively participate in motor-learning circuit rewiring by eliminating synapses in the striatum. The research identifies MEGF10 as a key molecular mediator of this process, which is regulated by dopamine signaling and neural activity.
Neural signals respond differently depending on time of day, with reduced activity at sunrise and enhanced at sunset. Blocking adenosine reveals a key regulator of cortical excitability across the day.
A small molecule called YM022 has been discovered to block aversive memory formation in mice, offering a new direction for developing anti-depressants. The study found that YM022 suppressed neuroplasticity-caused aversive memory formation and reduced depressive behaviors in mice.
The gene LRRC4 regulates synapse formation, stability and excitatory transmission, playing a crucial role in learning, memory formation and storage. It also has key implications in neuronal disorders and aggressive brain and spinal cord cancer.
Scientists discovered that an artificial cell membrane can exhibit long-term potentiation, a hallmark of biological learning and memory, persisting for many hours. This finding has the potential to revolutionize next-generation computing materials and architectures by merging functions of processing and memory in neuromorphic computers.
Scientists have developed a light-activated protein that can study single synapses in neurons, revolutionizing the understanding of long-term potentiation. The discovery reveals the physical changes in dendritic spines during long-term potentiation, providing valuable insights into learning and memory.
Researchers at Ruhr-University Bochum used EEG to study the impact of repetitive tactile stimulation on brain activity. They found that neuronal responses adapted to the frequency of stimulation and showed changes over time, potentially illustrating a learning process.
A research team led by Dr. Kea Joo Lee found that MAP2 plays a crucial role in inducing long-term potentiation, a cellular mechanism underlying learning and memory. The study's discovery provides key insights into synaptic plasticity mechanisms and potential therapeutic strategies for memory-related diseases.
Research reveals that Pannexin1 channel protein is critical for synaptic plasticity, a key process in learning and memory. Mice lacking Pannexin1 display autistic-like behavior and impaired spatial orientation, highlighting the importance of this channel for brain function.
Scientists at the Max Planck Institute of Psychiatry found that effective signal transmission in the hippocampus requires theta-frequency impulses, generating waves that propagate through the brain. This discovery explains why we are more productive after drinking coffee or experiencing stress.
Researchers at Duke University Medical Center have found a cascade of signaling molecules that allow brief signals to last for tens of minutes, forming stronger connections in the brain. This discovery could lead to new insights into diseases like Alzheimer's and autism.
Researchers at Max Planck Institute discover key molecule for LTP, a crucial process for learning and memory. The finding sparks new discussion on the role of LTP in memory formation, as mice lacking LTP showed no abnormal learning behavior.