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Study shows that human hearts generate new cells after birth

Researchers at Boston Children's Hospital found that young humans can generate new heart muscle cells, challenging the accepted wisdom on human heart growth. The study's findings offer a potential new approach to treating heart failure in children by stimulating cardiomyocyte proliferation.

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Healing heart attack victims, one cell at a time

Researchers have found that human heart cells develop into adulthood around age 6, with the percentage of new cells decreasing significantly with age. This discovery may lead to new pharmacological strategies to stimulate heart cell regeneration and complement cell transplantation.

Fruit fly research may lead to better understanding of human heart disease

Researchers have identified a connection between fruit fly genetics and human heart disease, revealing that certain genes play a role in both embryonic and adult heart function. The study found TBX20 mutations in humans with structural congenital heart abnormalities and heart muscle dysfunction, suggesting its potential involvement in ...

Protein overexpression at heart of heart failure

Increased expression of an accessory subunit of L-type calcium channels is linked to altered channel behavior in human heart failure. Researchers have identified a causal role for this protein overexpression in the development of cardiac dysfunction.

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Fruit fly's beating heart helps identify human heart disease genes

Researchers at Duke University Medical Center have developed a method to visualize the heartbeat of fruit flies, enabling them to identify genetic mutations associated with human heart disease. The team inserted a mutated gene into the fly genome and observed its effects, revealing similarities with human dilated cardiomyopathy.

Model simulates dynamics of heart rhythm disorders

A researcher created a model to simulate the dynamics of heart rhythm disorders, including ventricular tachycardia and fibrillation. The model revealed that only six spiral waves are present in the heart during fibrillation, contradicting previous assumptions about its chaotic nature.