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Cell Press


Understanding Fragile X syndrome with the blink of an eye

Fragile X syndrome is caused by a defect in the Fmr1 gene, which produces a nonfunctioning protein. Researchers found that mice lacking this gene only in specific neurons showed deficits in a motor learning task. The study also revealed abnormalities in signaling connections and dendrites of Purkinje cells in the cerebellum.

SourceCell Press·JournalNeuron·DateAug 3, 2005

Grasshopper love songs give insight into sensory tuning

Researchers found that grasshopper auditory neurons respond optimally to specific stimulus ensembles that differ from natural sounds but overlap with components of mating calls. This challenges the efficient coding hypothesis and suggests a weighted ensemble of natural stimuli based on behavioral relevance.

SourceCell Press·JournalNeuron·DateAug 3, 2005

Stem cells in bone marrow replenish mouse ovaries

A new study suggests that stem cells in bone marrow and blood can regenerate egg cells in adult mice, potentially leading to new fertility treatments and alternatives to hormone replacement therapy. The findings also provide evidence for the existence of germ cell progenitors outside of the ovary.

SourceCell Press·JournalCell·DateJul 27, 2005

Coral reef fish larvae settle close to home

Researchers tracked coral reef fish larvae using DNA paternity analysis and marking with tetracycline, finding that one third settled within a two-hectare area near their birth site. This study documents the smallest scale of dispersal for a marine fish species, providing insights into sustainable marine reserve management.

SourceCell Press·JournalCurrent Biology·DateJul 25, 2005

Blink, and the brain misses it

Researchers used a novel method to monitor brain activity during blinking, finding that brain areas responsible for visual input are temporarily suppressed. This neural mechanism may help prevent the brain from becoming aware of the eyelid sweeping down over the pupil during a blink.

SourceCell Press·JournalCurrent Biology·DateJul 25, 2005

Pinpointing a culprit molecule in Alzheimer's disease

Researchers created transgenic mice overproducing Ab40 and Ab42 to study their roles in AD pathology. The results showed that Ab42 mice accumulated extensive amyloid plaque and neural damage, while Ab40 mice showed little disease pathology. The study provides evidence for the critical role of Ab42 in initiating amyloid deposition.

SourceCell Press·JournalNeuron·DateJul 20, 2005

Insulin pulses keep the liver lean

New study reveals that acute insulin pulses limit fat synthesis in the liver by activating a key molecule called CEACAM1. Regular periods of fasting between meals help maintain a healthy liver by allowing insulin levels to fluctuate.

SourceCell Press·JournalCell Metabolism·DateJul 19, 2005

Secret sex life of killer fungus?

Researchers found nearly equal proportions of two different sexes in a worldwide collection of 290 specimens, indicating recent evolutionary history of sexual activity. This discovery could lead to new strains with increased ability to cause disease and infect humans.

SourceCell Press·JournalCurrent Biology·DateJul 11, 2005

How a baby's nose knows Mom's scent

Newborn rats' brains show decreased NMDA receptor activity in olfactory-deprived side, leading to increased neuronal activation and saliency of early odors. This critical period affects olfactory development and learning.

SourceCell Press·JournalNeuron·DateJul 6, 2005

Molecular trigger for Huntington's disease found

A study published in Neuron found that the abnormal HD protein selectively binds to and increases the level of p53 in cells, leading to increased cell death and mitochondrial dysfunction. This overactivation also causes behavioral abnormalities in mice engineered to have HD.

SourceCell Press·JournalNeuron·DateJul 6, 2005

Separating morning and evening in the circadian clock of mammals

Researchers found that cells in the caudal region of the suprachiasmatic nuclei (SCN) synchronize their gene-expression rhythms to dawn, while those in the rostral SCN exhibit an opposite response. This suggests that synchronous gene expression may be a hallmark of short-day acclimation, while regional de-synchrony increases on long days.

SourceCell Press·JournalCurrent Biology·DateJun 20, 2005

Placebos can bring emotional relief

Researchers found that placebo treatment reduced subjects' ratings of unpleasant pictures and activity in emotional centers. The study generalizes the concept of placebo effect and its underlying neural mechanisms for both pain perception and emotional stimuli.

SourceCell Press·JournalNeuron·DateJun 15, 2005

Studies reveal how cells sense oxygen

Research reveals that mitochondrial activity produces reactive oxygen species that signal low oxygen levels, allowing cells to adapt and respond. Cells with disabled mitochondria fail to detect changes in oxygen availability, highlighting the critical role of mitochondria in oxygen sensing.

SourceCell Press·JournalCell Metabolism·DateJun 7, 2005

Link between mothers' poor diets, kids' obesity

A study published in Cell Metabolism suggests that premature leptin surge in newborn mice of underfed mothers leads to remodeling of key brain circuits contributing to obesity later in life. The researchers found that the early leptin surge alone causes accelerated weight gain.

SourceCell Press·JournalCell Metabolism·DateJun 7, 2005

Pathogens shape evolution of humans' most diverse genes

A new study reveals that pathogens play a significant role in shaping the evolution of human's most diverse genes, specifically those encoding Human Leukocyte Antigen (HLA) proteins. High HLA diversity is found to correlate with high pathogen diversity, particularly for certain types of HLA genes.

SourceCell Press·JournalCurrent Biology·DateJun 6, 2005

Opiate cocktail may spare cells from morphine's dark side

Researchers developed a drug cocktail combining morphine with methadone to prevent cellular signaling imbalances associated with opiate tolerance. The study found that rats receiving the cocktail experienced reduced morphine dependence, suggesting a potential approach for treating chronic pain.

SourceCell Press·JournalCurrent Biology·DateJun 6, 2005

Where life's memories are stored

Studies of brain-damaged patients reveal that remote autobiographical memories can be preserved even with limited damage to the medial temporal lobe. The researchers found that the ability to recall and experience these memories is more closely tied to neocortical areas than previously thought.

SourceCell Press·JournalNeuron·DateJun 1, 2005

Exploring the 'corner-of-your-eye' phenomenon

The study found that when subjects focus on one feature, they also attend to similar features elsewhere in the visual field. The researchers developed an experimental technique to measure this phenomenon and its implications for understanding how our brain processes visual information.

SourceCell Press·JournalNeuron·DateJun 1, 2005

After a time-shift, mixed signals from the circadian clock

Researchers discovered that rat brains exhibit distinct responses to a six-hour delay in the light schedule, with one region adapting quickly and another taking several days. The study suggests that this difference in response is due to the neurotransmitter GABA, which affects brain regions differently.

SourceCell Press·JournalCurrent Biology·DateMay 23, 2005

Discovery of a 'drug anticipation' brain signal

Scientists detect dopamine release from nucleus accumbens in rats, revealing three distinct signals associated with cocaine administration. The findings suggest that dopamine release transients are highly plastic and governed by motivation, learned association, and pharmacological actions.

SourceCell Press·JournalNeuron·DateMay 18, 2005

Words influence smells

Subjects rated odors more pleasant when labeled with positive words like 'cheddar cheese', whereas unpleasant labels had the opposite effect. The study also found that brain regions processing odors were activated by word labels, highlighting high-level cognitive inputs' impact on sensory perception.

SourceCell Press·JournalNeuron·DateMay 18, 2005

Fat's fate depends on its source

A new study published in Cell Metabolism found that normal metabolism requires so-called "new" fat to maintain glucose, lipid, and cholesterol homeostasis. The researchers discovered that old fat stores alone failed to set critical metabolic pathways in motion, leading to a marked decline in gene activity.

SourceCell Press·JournalCell Metabolism·DateMay 10, 2005

Excess liver gene protects against high-fat diet

A new study reveals that mice with an excess of liver X receptor (LXRa) are protected from high-fat diet-induced cholesterol and blood vessel plaques. The findings suggest that natural variation in LXRa levels can influence susceptibility to heart disease, depending on diet.

SourceCell Press·JournalCell Metabolism·DateMay 10, 2005