Researchers at Johns Hopkins Medicine have made a groundbreaking discovery that prostaglandin-E2 (PGE2) protects brain cells from damage caused by stroke. The finding provides a new strategy for tackling and understanding the condition, which affects 4 million Americans annually.
A study of 213 premature babies found that brain damage was most likely caused by prematurity and infections. The research revealed that even with advances in medical care, such as electronic fetal heart rate monitoring, the incidence of cerebral palsy and other problems has remained unchanged.
A new device called SARA has been developed to detect fetal brain activity using magnetoencephalography (MEG) and light stimulation. The study found that 4 of 10 fetuses had measurable brain responses to light pulses, with the response time decreasing with increasing gestational age.
A new technique uses cognitive testing to locate vital brain vessels, allowing for safer embolization procedures. In a study of 14 patients, 12 showed no significant neurological damage after targeted vessel treatment, while 2 required alternative approaches.
A study found that prolonged abstinence from alcohol can lead to partial reversal of structural brain damage, particularly in the frontal lobes and cerebellum. This suggests that brain function can improve with abstinence, offering hope for recovering alcoholics.
A study published in Nature found that individuals with aphasia, who experience language loss after a stroke or brain damage, possess an exceptional ability to detect deception. They were able to identify lies cued by facial expressions alone with high accuracy, outperforming other groups.
A recent study published in Alcoholism: Clinical & Experimental Research found that short-term alcoholics demonstrate the same brain damage as long-term alcoholics. The study suggests that there may be separate vulnerabilities to developing an alcohol dependency and brain damage, and that recovery from brain damage can take time.
A new Johns Hopkins study shows that ecstasy users are at risk of developing brain injury due to the drug's impact on serotonin-releasing nerves. The research found significant losses in these nerve cells with greater use, suggesting potential long-term damage.
A new study suggests that a drug that inhibits nitric oxide production may reduce the risk of brain damage during heart surgery. The study found that dogs treated with the drug showed significantly less apoptotic brain cell damage and decreased nitric oxide in the brain compared to untreated dogs.