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DNL343 and disease progression in amyotrophic lateral sclerosis

09.22.26 | JAMA Network

About the Study: In this randomized clinical trial, despite the relevance of the eIF2b activation pathway in ALS disease biology and supporting evidence of proof of mechanism and optimal dose selection in a prior phase 1B ALS clinical trial, 200-mg/d DNL343 did not show evidence of slowing disease progression in ALS, highlighting the need for alternative therapeutic approaches.

Corresponding Author : Sabrina Paganoni, MD, PhD, Sean M. Healey and AMG Center for ALS at Massachusetts General Hospital, 165 Cambridge St, Ste 600, Boston, MA 02114 ( spaganoni@mgh.harvard.edu ).

10.1001/jamanetworkopen.2026.34809

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How to Cite This Article

APA:
JAMA Network. (2026, September 22). DNL343 and disease progression in amyotrophic lateral sclerosis. Brightsurf News. https://www.brightsurf.com/news/LVDOD2YL/dnl343-and-disease-progression-in-amyotrophic-lateral-sclerosis.html
MLA:
"DNL343 and disease progression in amyotrophic lateral sclerosis." Brightsurf News, Sep. 22 2026, https://www.brightsurf.com/news/LVDOD2YL/dnl343-and-disease-progression-in-amyotrophic-lateral-sclerosis.html.