A study published in the European Heart Journal found that male bodybuilders have a high risk of sudden cardiac death, with professional bodybuilders facing a more than fivefold increase in risk. The study highlights the need for greater awareness, preventive strategies and policy changes within the bodybuilding community.
A study published in Science reveals that ganglia in the neck region are responsible for disrupting melatonin production and causing sleep disturbances in people with heart conditions. Researchers found that macrophages accumulate in the ganglion, leading to inflammation and scarring, which can be treated with drugs.
A study found that firefighters who died from cardiac arrest were more likely to have narrowed arteries, an enlarged heart, and increased wall thickness of the left ventricle. These conditions increase the risk of death from cardiac arrest, highlighting the importance of medical screening for firefighters.
Researchers at Loyola University Chicago Stritch School of Medicine have identified a regulatory protein that binds to RSK, keeping its activity in check. This discovery could lead to new treatments for breast cancer, prostate cancer, and heart enlargement, as well as Carney complex.
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Researchers have identified genes that can cause enlarged hearts and kidney inflammation, leading to heart attacks and kidney failure. Understanding these genetic links may lead to new treatments for common heart diseases.
Researchers identified a key step in cocaine-induced heart enlargement, where high concentrations of the drug lead to increased calcium within heart muscle cells, prompting the accumulation of primitive fetal proteins. This process can cause irregular heart rhythms and sudden death, affecting young people between 18-29 years old.
Researchers at Johns Hopkins Medicine discovered that altering leptin's signaling pathway can minimize or even reverse left ventricular hypertrophy, a condition causing the heart to expand and stiffen. Leptin infusions led to weight loss and improved heart function in mice with obesity, while caloric restriction had no effect.
Researchers at UT Southwestern Medical Center have identified a gene, HDAC9, that limits abnormal heart-muscle growth. The study shows that HDAC9 restricts cardiac growth in response to stress but does not affect normal growth during development or exercise.
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