Articles tagged with Brain Ischemia
Researchers found that brain-derived neurotrophic factor suppressed glucose intolerance by improving insulin receptor expression and inhibiting gluconeogenesis in the liver. This suggests a potential therapeutic hypothesis for treating glucose intolerance after cerebral ischemia.
The study reveals rutaecarpine's ability to improve neurological function after cerebral ischemia reperfusion injury. The mechanism involves reducing oxidative stress, providing a promising basis for rutaecarpine as an ideal drug for preventing and treating cerebral ischemia reperfusion injury.
Research found that 60-minute sevoflurane preconditioning reduces infarct volume and apoptotic cells, but longer durations do not provide additional benefits. The study suggests that shorter preconditioning periods may be more effective in protecting against cerebral ischemia.
Researchers found that cilostazol decreases hypoxia-inducible factor-1α and heme oxygenase-1 expression levels, protecting against cognitive impairment induced by chronic cerebral ischemic injury. Cilostazol treatment also exhibits an anti-apoptotic mechanism in the context of chronic cerebral ischemia.
The motor relearning program significantly promotes neuronal regeneration and repair, as well as angiogenesis in damaged brain tissue. This study provides a new theoretical idea for the clinical treatment of brain ischemia.