Add BrightSurf on Google Email

Heidelberg University Hospital


Protection from severe malaria explained

Researchers discovered that a degradation product of defective hemoglobin blocks the establishment of a trafficking system used by the malaria parasite, preventing circulatory disorders and neurological complications. The study provides new insights into the molecular mechanism behind the protective effect of sickle-cell anemia against...

SourceHeidelberg University Hospital·JournalScience·DateNov 18, 2011

How nerve cells distinguish odors

Researchers at Heidelberg University Hospital discovered that inhibiting communication between nerve cells accelerates odor discrimination in mice. The study confirmed the function of lateral inhibition, which enables quick and reliable recognition of similar smells, using genetic manipulation and electrophysiological measurements.

Communication problems in the brain

A study published in PNAS found that a synaptic maturation disorder, particularly with the neuroligin-1 protein, may trigger autism. The research suggests that an insufficient amount of this protein can impair the maturation process at nerve terminals, leading to communication problems and weaknesses in social contacts.

SourceHeidelberg University Hospital·JournalProceedings of the National Academy of Sciences·DateJan 20, 2010

Old stain in a new combination

Researchers from Heidelberg University Hospital have discovered that combining methylene blue with newer malaria medications is twice as effective in curbing the spread of malaria parasites than standard therapy. This new approach has significant implications for eradicating the disease, particularly among children under five who are m...

SourceHeidelberg University Hospital·JournalPLOS ONE·DateMay 20, 2009

How tumor cells move

Scientists at Heidelberg University Hospital have identified a previously unknown protein called SCAI that inhibits the movement and spread of tumor cells. The study suggests that SCAI could be an interesting starting point for research into new mechanisms for fighting cancer.

SourceHeidelberg University Hospital·JournalNature Cell Biology·DateApr 8, 2009

Why 'lazy Susan' has a weak heart

Heidelberg cardiologists have discovered a single amino acid mutation that can severely limit heart function in zebrafish, a model similar to the human genome. The researchers hope to develop new therapies for patients with cardiac insufficiency by targeting this mutation.

SourceHeidelberg University Hospital·JournalCirculation Research·DateMar 5, 2009

Enzyme weakens the heart

Researchers from Heidelberg University Hospital found that CaMKII delta enzyme activates stress response and hypertrophy of the heart. Genetic suppression of this enzyme protects mice from chronic cardiac insufficiency, offering a promising approach for systematic prevention.

SourceHeidelberg University Hospital·JournalProceedings of the National Academy of Sciences·DateFeb 17, 2009

Tiny details in three dimensions

Heidelberg researchers use cryo-electron tomography to visualize the structure of Lyme disease bacteria, revealing differences in motility systems that may explain varying symptoms between European and North American patients. The study provides new insights into the complex biology of borreliosis and potential therapeutic targets.

SourceHeidelberg University Hospital·JournalMolecular Microbiology·DateFeb 13, 2009

Hope for preventative treatment for cystic fibrosis lung disease

Researchers have successfully prevented cystic fibrosis lung disease in a mouse model by administering amiloride, a drug that inhibits hyperactive sodium channels. Early inhalation treatment prevents chronic lung damage and airway inflammation, providing a new therapeutic approach for the widespread genetic disease.

SourceHeidelberg University Hospital·JournalAmerican Journal of Respiratory and Critical Care Medicine·DateFeb 5, 2009