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Stanford Medicine


Disruption of immune-system pathway key step in cancer progression, Stanford study shows

A Stanford University study found that disrupting the interferon pathway, a critical immune signaling mechanism, can lead to cancer progression and impaired immune response. This defect was detected in patients with breast, melanoma, and gastrointestinal cancers, suggesting a possible explanation for treatment failures.

SourceStanford Medicine·JournalProceedings of the National Academy of Sciences·DateMay 18, 2009

Sex is in the brain, says new research from Stanford

A new study from Stanford University School of Medicine found that brain activity patterns differ in women with hypoactive sexual desire disorder compared to those without the condition. The research suggests that increased attention to one's own responses to erotic stimuli may play a role in sexual dysfunction.

SourceStanford Medicine·JournalNeuroscience·DateMar 2, 2009

Protein complex shown to play pivotal role in stem cell development in 2 Stanford studies

Scientists at Stanford University School of Medicine have identified a protein complex that plays a pivotal role in controlling the ability of embryonic stem cells to become any cell type. The finding is an important advance in harnessing the unique abilities of embryonic stem cells to treat disease and generate replacement tissue.

SourceStanford Medicine·JournalProceedings of the National Academy of Sciences·DateMar 2, 2009

Immune system 'atlas' will speed detection of kidney transplant

A new immune-system 'atlas' will improve doctors' ability to monitor transplanted organs and shed light on the mechanisms of gradual, cumulative kidney malfunction after transplant. The atlas allows for inexpensive, noninvasive blood tests that show whether a donated kidney is infected or accruing chronic injuries.

SourceStanford Medicine·JournalProceedings of the National Academy of Sciences·DateFeb 23, 2009

Cell movements totally modular, Stanford study shows

Researchers at Stanford University School of Medicine have shown that distinct groups of proteins each control one of four simple activities involved in the cells' collective migration. The study overturns an assumption common in genomics and provides a powerful tool for developing new therapeutics.

SourceStanford Medicine·JournalGenes & Development·DateNov 30, 2008