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Cincinnati Children's Hospital Medical Center


Advancing the understanding of an understudied food allergy disorder

Eosinophilic gastritis is a chronic inflammatory condition characterized by high levels of eosinophils in the blood and gastrointestinal tract. The study found that it involves allergy-associated-immune mechanisms and has a distinct gene expression pattern, paving the way for improved diagnosis and treatment.

SourceCincinnati Children's Hospital Medical Center·JournalJournal of Allergy and Clinical Immunology·DateSep 23, 2014

Secondhand smoke exposure increases odds of hospital asthma readmission for children

A new study finds that exposure to secondhand smoke at home or in the car increases the odds of children being readmitted to the hospital within a year of being admitted for asthma. Cotinine levels measured in the blood and saliva of over 600 children showed a significant correlation between tobacco exposure and increased risk of futur...

Protein explains increased asthma severity in children exposed to diesel exhaust from traffic

A new study published by Cincinnati Children's Hospital Medical Center found a strong link between exposure to diesel exhaust and increased asthma severity in children. The research suggests that neutralizing the protein IL-17A could be a useful potential therapeutic strategy to counteract the effects of traffic-related air pollution.

SourceCincinnati Children's Hospital Medical Center·JournalJournal of Allergy and Clinical Immunology·DateSep 23, 2013

Study suggests antioxidant treatment may help NF1-linked behavioral issues

Researchers found that defects in the NF1/Ras molecular pathway trigger production of harmful oxidative nitric oxide molecules in glial brain cells, leading to behavioral issues. Antioxidant treatment reversed symptoms in mouse models of NF1 and Costello syndrome, suggesting a potential therapeutic approach for related human conditions.

Research uncovers potential preventive for central line infection

Researchers at Cincinnati Children's Hospital Medical Center have developed an antibody that prevents Candida albicans from binding to heparin, stopping biofilm formation in a rat model. The study suggests that standard anticoagulants may facilitate biofilm formation by microbes, highlighting the need for new prevention strategies.

SourceCincinnati Children's Hospital Medical Center·JournalThe Journal of Infectious Diseases·DateSep 11, 2013