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Stanford Medicine


DNA damage response links short telomeres, heart disorder in Duchenne muscular dystrophy

A study by researchers at Stanford University School of Medicine found that progressively shortening telomeres in heart muscle cells triggered a DNA damage response compromising mitochondrial function. This led to cardiomyopathy and death in mice with Duchenne muscular dystrophy, suggesting new therapeutic targets.

SourceStanford Medicine·JournalProceedings of the National Academy of Sciences·DateOct 31, 2016

Possible psoriasis drug target identified by Stanford researchers

Researchers at Stanford University School of Medicine have identified Rac1 as a potential target for treating psoriasis. The small protein is involved in wound repair and has been linked to environmental triggers of the disease, as well as genetic predisposition. Blocking Rac1 activation may offer a new way to treat psoriasis without s...

SourceStanford Medicine·JournalJournal of Clinical Investigation·DateJun 13, 2016