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1,000+ results for "Pathology"

Leukemic cells find safe haven in bone marrow

Research at St. Jude Children's Research Hospital shows that leukemic cells receive a protective niche from bone marrow mesenchymal cells, which release the amino acid asparagine to help them survive treatment with asparaginase. This finding suggests new strategies for enhancing anti-leukemic drugs and reducing leukemia recurrence rates.

SourceSt. Jude Children's Research Hospital·JournalJournal of Clinical Investigation·DateMar 22, 2007

Treating male infertility with stem cells

Researchers transplanted bone marrow stem cells into testes of infertile mice, showing potential for treating male infertility. The stem cells differentiated into germ cells and supporting cells, suggesting a new approach to replacing nonfunctioning cells involved in sperm production.

SourceAmerican Journal of Pathology·JournalAmerican Journal Of Pathology·DateMar 1, 2007

Irish potato famine disease came from South America

Researchers at North Carolina State University have found that Phytophthora infestans, the fungus-like pathogen responsible for the 1840s Irish potato famine, originated in the Andes region of South America. The study used gene genealogies to track the migration patterns of different strains of the pathogen, pointing to an Andean origin.

SourceNorth Carolina State University·JournalProceedings of the National Academy of Sciences·DateMar 1, 2007

Prion disease treatable if caught early

Researchers found that early brain degeneration can be reversed if prions are depleted in neurons, leading to improved cognitive function and reversal of neurological pathology. This discovery opens new avenues for targeting neuronal prion protein as a therapeutic approach and may enable early intervention in human prion disease.

SourceCell Press·JournalNeuron·DateJan 31, 2007

Transdermal vaccine effective in treating Alzheimer's disease in mouse model

Researchers have developed a novel transdermal vaccine approach that effectively clears brain-damaging plaques from a mouse model of Alzheimer's disease. The vaccine works by triggering the immune system to recognize abnormal protein Ab as a foreign invader, reducing cognitive deficits and senile plaque burden.

SourceUniversity of South Florida (USF Health)·JournalProceedings of the National Academy of Sciences·DateJan 22, 2007

A new jump start for aging blood vessels

Researchers found that inactivating TNFa significantly reduced blood vessel impairment and cell death in aged vessels, suggesting a link between increased TNFa levels and cardiovascular disease. The study's findings could lead to the development of new treatments for age-related vascular diseases.

SourceAmerican Journal of Pathology·JournalAmerican Journal Of Pathology·DateDec 28, 2006

'Muscle' protein drives prostate cancer

Researchers at Johns Hopkins Kimmel Cancer Center found overproduction of myosin VI in prostate tumor cells and precancerous lesions. Silencing myosin VI in lab studies reduced cell invasion, suggesting its critical role in starting and maintaining malignant properties of most human prostate cancers.

SourceJohns Hopkins Medicine·JournalAmerican Journal Of Pathology·DateNov 8, 2006

Saving salivary glands from the collateral damage of radiation therapy

Targeted overexpression of HSP25 protects salivary gland function by maintaining gland weight, salivary flow rate, and salivary fluid composition. HSP25 also preserves expression of aquaporin 5, crucial for water transport in salivary glands. These findings suggest a novel radioprotective strategy against radiation-induced salivary gla...

SourceAmerican Journal of Pathology·JournalAmerican Journal Of Pathology·DateOct 27, 2006

How Rett Syndrome mutation targets the brain

Researchers identified a key mechanism behind Rett Syndrome by pinpointing the S421 site on the MeCP2 protein responsible for its normal function. This specificity explains why mutations affecting that site target brain development, leading to delays in motor skills and speech loss.

SourceCell Press·JournalNeuron·DateOct 18, 2006

Untangling a pathology of Alzheimer's

Researchers have identified an enzyme called puromycin-sensitive aminopeptidase (PSA) that snips apart abnormal protein tangles in the brain, potentially providing a protective mechanism against neurodegeneration. Higher levels of PSA gene expression were found in cerebellum regions resistant to neurodegeneration.

SourceCell Press·JournalNeuron·DateSep 6, 2006

Onchocerciasis treatment reduces prevalence and intensity by 38 percent

Researchers from Hospital Clínic de Barcelona found that a 8-year Ivermectin treatment reduced onchocerciasis prevalence by 38.4%, while incidence of disease decreased by 2.6 times in children aged 0-4. The study, financed by the Spanish Agency and WHO, marks a significant step forward in combating river blindness.

SourceIDIBAPS - Institut d'Investigacions Biomèdiques August Pi i Sunyer·JournalTropical Medicine & International Health·DateJul 28, 2006

Free radical cell death switch identified

Researchers at Harvard Medical School have defined a molecular signaling pathway linking oxidative stress to cell death in brain neurons. The discovery of the MST-FOXO biochemical switch mechanism may provide potential new targets for the diagnosis and treatment of age-related diseases such as Alzheimer's.

SourceHarvard Medical School·JournalCell·DateJun 1, 2006

Combined contrast enhanced MRI shows promise

A new study reveals that combined contrast enhanced MRI (CCE-MRI) closely matches the staging of non-alcoholic fatty liver disease (NAFLD) using conventional pathology. CCE-MRI offers a safer alternative to invasive liver biopsy, directly visualizing fibrosis and detecting liver cancers simultaneously.

SourceAmerican College of Radiology·JournalAmerican Journal of Roentgenology·DateMay 1, 2006

Mapping alcohol brain damage

Associate Professor Peter Dodd's team is using microarray analysis to study brain tissue from dead alcoholics, aiming to identify genes responsible for the damage. The research could lead to a better understanding of neurological diseases and potentially develop a drug to reduce alcohol dependence.