A new study by University of Wisconsin-Madison biochemist Ron Raines found that a ribonuclease A protein in humans has the same cancer-fighting potential as a frog-derived protein. The finding opens a door to creating a new class of natural drugs aimed at fighting cancer without side effects.
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Researchers found cancer cells self-destruct when glucose is cut off, suggesting a potent new way to fight cancer with few side effects. The discovery was made using a compound that disrupts glycolysis, the process that produces energy from sugar, and could be used in combination with existing treatments.
Researchers at Case Western Reserve University have discovered that epigallocatechin-3-gallate, a green tea compound, can induce programmed cell death in cancer cells without harming healthy cells. This finding offers new hope for cancer prevention and treatment, and may lead to the development of purified polyphenolic derivatives.
Researchers are developing a new approach to cancer treatment called adoptive immunotherapy, which uses tumour antigens to stimulate immune cells. Tumour antigens are identified and used to sensitize T cells, which then recognize and attack cancer cells.
Researchers have discovered compounds in the bark of the pawpaw tree that show preliminary success in fighting some drug-resistant cancers. The compounds, called Annonaceous acetogenins, preferentially kill multidrug-resistant cells by inhibiting ATP production, pulling the plug on their energy source.
A team of physicians from Memorial Sloan-Kettering Cancer Center and the University of Rochester's Cancer Center have developed a herpes-based gene therapy that recruits the immune system to target and eliminate cancer cells in the liver. The vaccine has shown promising results, with laboratory rats achieving complete remission and few...
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A promising hepatitis-based gene therapy has been developed to target and eliminate liver cancer cells. The treatment, created by a team of physicians, recruits the body's immune system to attack and wipe out cancerous tumors.
Cancerous cells produce high amounts of free-radicals that promote uncontrolled cell growth; antioxidants and protein inhibitors can suppress this process, slowing down runaway cell division.
Researchers have identified an inactive gene that contributes to cancer development when it fails to work in harmony with the tumor-suppressing p53 gene. The findings suggest that the bax gene can act as a tumor suppresser, and its dysfunction may lead to increased cancer cell death and tumor growth.