Articles tagged with Memory Disorders
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Researchers found that DBS in either brain target effectively treated motor symptoms, but produced unique effects on mood and mental sharpness. The discovery may impact the selection of DBS patients, especially those with pre-existing memory or cognitive disabilities.
Researchers at Wellcome Trust Centre for Neuroimaging discovered that brain activity can be used to predict spatial memories and locations. The study's findings challenge current scientific thinking and offer insights into how memories are encoded across neurons.
A team of Dutch researchers successfully reduced the fear response in human volunteers using beta-blocker propranolol. The treatment weakened fear memories, allowing them to be erased from long-term memory. This breakthrough has implications for treating anxiety disorders and offers a new approach to intervention.
Researchers at Boston University School of Medicine found that people whose parents have Alzheimer's disease or dementia perform worse on formal memory testing. The study also identified a strong genetic link between ApoE ε4 gene and the transmission of Alzheimer's disease risk between generations.
A recent study found that individuals with parents diagnosed with Alzheimer's disease or dementia may experience low verbal and visual memory performances in middle age. The study suggests that the ApoEe4 gene plays a role in the transmission of Alzheimer's risk between generations.
A study of 197 people with mild cognitive impairment found that engaging in mental activities like reading, crafting, and playing games reduced the risk of memory loss by 30-50% compared to those who didn't participate. Participants who watched TV for less than 7 hours a day were also 50% less likely to develop memory loss.
Researchers at VIB confirm GPR3 protein's role in reducing amyloid plaques, a hallmark of Alzheimer's disease. The discovery paves the way for developing a new medicine to combat the disease and preserve brain cells.
Researchers found a statistically significant inverse relationship between A1C levels and cognitive test scores in people with type 2 diabetes. The study suggests that poorer blood glucose control is strongly associated with poorer memory function.
Researchers found that BDNF treatment can prevent or reverse memory loss, cognitive impairment, and brain cell degeneration in animal models of Alzheimer's disease. The study suggests a potential novel approach to treating the disease by slowing its progression.
A new study published by Elsevier found that nearly half of patients with heart failure have problems with memory and other aspects of cognitive functioning. Memory problems, especially short-term memory, were the most common type of cognitive deficit in these patients.
A new study published in Neurology, a journal of the American Academy of Neurology, found that higher education levels do not slow down the rate of memory loss. Despite initial improvements in memory and thinking skills with more education, these advantages did not translate to slower decline over time.
Researchers found that insulin shields neurons from toxic proteins in Alzheimer's disease, preserving memory-forming synapses. This discovery suggests a novel approach to treating the devastating disease by increasing insulin sensitivity in the brain.
A drug used to treat stroke patients may also improve learning and memory and reduce the risk of Alzheimer's disease, according to a new study. The active form of the parent drug Fasudil improved spatial learning and working memory in middle-aged rats.
A study published in Cortex found that reducing attentional resources enhances memory accuracy in confabulating patients. By dividing their attention between tasks, these patients showed significantly reduced false-memory levels.
A new study by UNC researchers found that postmenopausal women with hypoactive sexual desire disorder (HSDD) report poorer health status, worse quality of life, and physical symptoms such as back pain and memory problems compared to those without the disorder.
The DREAM gene plays a key role in regulating pain perception, learning, and memory. Mice without the gene show reduced sensitivity to pain but improved memory, suggesting a link between calcium regulation and brain function.
Two new studies link hormone therapy to reduced brain volume in critical memory areas, accelerating loss of cognitive function in postmenopausal women. Researchers found no correlation between hormone therapy and increased brain lesions, contradicting previous hypotheses.
New studies have found a link between hormone therapy and brain shrinkage in postmenopausal women, with smaller brain volumes in areas involved in thinking and memory skills. However, hormone therapy was not linked to an increase in small vascular lesions or 'silent strokes' that are often the first sign of cerebrovascular disease.
Researchers found MRI brain scans accurately diagnose Alzheimer's disease even before symptoms interfere with daily function. The study suggests incorporating MRIs into the assessment of patients with memory problems can standardize and improve early diagnosis.
Researchers found that older adults remember fewer negative events than younger counterparts due to differences in brain activity. The study suggests that seniors' brains use thinking rather than feeling processes to store emotional memories, which may improve understanding of mental health issues like depression and anxiety.
Decreasing FKBP12 gene activity in mice disrupted neuron-to-neuron communication, leading to enhanced long-term potentiation and repetitive behaviors. The study provides insight into the molecular mechanisms underlying neurodevelopmental disorders such as autism spectrum disorder and obsessive-compulsive disease.
A study reveals a link between dysregulation of a common signaling pathway and repetitive behaviors associated with multiple neurological disorders. Deletion of FK506-binding protein is associated with enhanced mTOR signaling, synaptic plasticity, and memory deficits.
Researchers used fMRI to find that patients with stress-related psychiatric disorders have abnormal brain activation in the prefrontal cortex and hippocampus, affecting memory suppression. This can lead to poor memory and increased anxiety due to lingering traumatic memories.
A large clinical trial found that ginkgo biloba has no effect on the onset of dementia or development of Alzheimer's disease. The study tested 120 milligrams of ginkgo twice daily versus placebo in 3,069 participants and showed that it had no impact on cognitive decline or mortality.
Researchers at Northwestern University's Feinberg School of Medicine discovered that super-aged brains have a lower number of fiber-like tangles, which accumulate inside brain cells and are thought to kill them. This finding suggests that individuals with fewer tangles perform better cognitively.
A Baylor College of Medicine neuroscientist has identified a single molecule that can affect memory, with findings showing that reducing activity of a key protein increases the expression of genes and proteins needed for long-lasting memories. The study also reveals that short exposure to experiences creates long-term memories.
Researchers found that valproic acid improved memory and reduced brain lesions in mice with an AD-like disease. The drug also prompted damaged nerves to start repairing themselves and improved performance in memory-related tests.
Researchers found two patients with hippocampal damage showed strikingly different patterns of memory performance, one with no deficit and the other impaired. The study suggests that subtle differences in brain pathology may be critical to understanding memory outcomes.
A study published in Neurology found that people with more education and mentally demanding occupations had significantly less brain damage from Alzheimer's disease than those with less education and less demanding jobs. Higher education was associated with a stronger brain reserve, allowing it to compensate for damage and maintain fun...
Researchers discovered specific fatty acids contributing to Alzheimer's disease progression and proposed a therapeutic strategy by inhibiting an enzyme regulating fatty acid levels. Lowering the enzyme's activity improved cognitive deficits in mouse models.
A new study suggests that people with Alzheimer's disease may have lower blood pressure because of cognitive decline. Failing memory causes forgetfulness, which can lead to a decrease in stress-related high blood pressure.
A study of 500 people aged 50-85 found a link between occasional memory lapses and smaller hippocampus volumes. Researchers discovered that these subjective memory complaints were associated with reduced brain volumes, potentially indicating early signs of cognitive decline.
A TGen-led team found a link between the brain protein KIBRA and reduced Alzheimer's disease risk in individuals with the T-allele gene. The study suggests that KIBRA may play a role in Alzheimer's disease, providing promising new targets for treatments.
Research reveals that as we age, our brains struggle to ignore irrelevant information and process information rapidly, leading to decreased memory formation. This breakdown occurs early in visual processing and persists despite attempts to suppress extraneous information.
Research reveals that chronic alcohol consumption alters neural systems in the brain, leading to cognitive deficits and disorders of memory and learning. Dynorphins, a key player in this process, are found to be upregulated in the prefrontal cortex and hippocampus, impairing learning and memory.
Researchers have discovered a new process that regulates adverse responses to bad memories by working on a tiny group of neurons inside the amygdala. By blocking or activating specific receptors, scientists can speed up or slow down traumatic responses, offering potential new treatments for panic disorders.
A small sample of adults with mild age-related memory loss showed increased brain metabolism and normalized brain function after taking Aricept for 18 months. The study suggests treating early symptoms of memory loss may protect the brain and help with mild age-related cognitive decline.
A study published in Neurology found that statin users were about half as likely to develop dementia as non-users. Researchers analyzed data from 1,674 Mexican Americans over 60 years old and found a significant association between statin use and reduced risk of cognitive decline.
Researchers found a significant association between mild memory impairment and impaired central auditory processing. Individuals with mild memory impairment showed lower scores on central auditory processing tests, indicating difficulty managing competing signals.
Researchers at Rutgers University have identified a key component of the amygdala's neural network involved in extinction, or elimination, of fear memories. Clusters of intercalated neurons inhibit amygdala outputs to brain stem structures generating fear responses, offering potential for pharmacological interventions.
A UCLA professor's analysis of over 160 studies reveals that a balanced diet rich in omega-3 fatty acids can enhance cognitive abilities, protect the brain from damage, and counteract aging. Omega-3s have been shown to improve learning and memory, fight depression and mood disorders, and even support synaptic plasticity.
A study from MIT researchers found that a dietary cocktail combining omega-3 fatty acid, uridine, and choline improves memory and learning in gerbils. The combination is being tested in Alzheimer's patients, suggesting a possible beverage-based treatment for the disease.
Researchers identify key role of endogenous opioids in alcoholism-related cognitive impairment. Innovative therapies may improve or prevent alcohol-induced memory problems in patients with alcohol dependence.
Researchers discovered that calpain inhibitors restore normal signaling between nerve cells, improving memory in APP/PS1 mice. Calpain inhibition may slow down or stop Alzheimer's disease progression by addressing impaired cognition.
Researchers from the American Heart Association report a link between low levels of high-density lipoproteins (HDL) and increased risk of memory loss and dementia. Studies found that participants with low HDL cholesterol showed a higher risk of memory decline compared to those with high HDL, particularly in late midlife.
Adults with type 2 diabetes who eat high-fat meals may experience immediate memory declines, but taking antioxidant vitamins with the meal can offset this effect. A study found that consuming healthy foods rich in fruits and vegetables is the best defense against cognitive decline.
Researchers analyzed brain activity using fMRI to identify differences in connectivity between Alzheimer's patients and healthy individuals. They found that patients had fewer functional hubs, compromising regional connections and contributing to memory loss.
Researchers at UCLA discovered that rapamycin reverses learning deficits caused by tuberous sclerosis complex (TSC), a genetic disorder also linked to autism. The study shows that the disease's impact can be reversed through biochemical changes, restoring normal brain function and memory.
O'Keefe's discovery of place cells revolutionized the field of cognitive neuroscience, enabling animals to navigate complex environments. His research has also shed light on Alzheimer's disease, revealing a deterioration in spatial information content in aged mice.
Researchers at UCLA have discovered a link between sleep apnea and memory loss, finding nearly 20% smaller mammillary bodies in patients with the disorder. This suggests a long-lasting brain injury from repeated drops in oxygen, leading to cell death and inflammation.
A recent meta-analysis found that smoking is associated with increased risk of poor memory among middle-age adults. Long-term ex-smokers also showed improvement in other health behaviors after quitting smoking.
A pilot study showed that older adults with early memory decline who drank Concord grape juice daily experienced improved list learning and trends of short-term retention and spatial memory. The study, led by Robert Krikorian, found significant benefits in cognitive function.
A randomized controlled trial found that exposure therapy was more effective than cognitive restructuring in preventing PTSD in individuals with acute stress disorder. Exposure therapy reduced distress ratings and promoted full remission, suggesting its potential as an early intervention for high-risk patients.
A recent Harvard study found that individuals with an active social life may experience a slower rate of memory decline compared to those with lower levels of social integration. The study, which analyzed data from over 1,000 US adults aged 50 and older, suggests that strong social networks can help preserve memory function.
Researchers from the University of Bristol have identified a key molecular mechanism controlling synaptic plasticity, which is vital for visual recognition memory and learning. Blocking this mechanism prevents visual recognition memory in rats, demonstrating its importance in brain function.
A new study reveals that protein-destroying machines in nerve cells play a crucial role in how memories are formed. The researchers found that blocking activity in these machines could potentially strengthen synapses and improve memory, offering new hope for treating Alzheimer's and other brain diseases.
A new study by Deniz Erten-Lyons shows that a larger hippocampus in the brain may protect individuals from the effects of Alzheimer's disease. Researchers compared the brains of 12 people with sharp memories to those of 23 individuals diagnosed with Alzheimer's disease, finding a significant difference in hippocampus volume
Researchers found that phytochemical-rich foods like blueberries improve spatial working memory tasks within three weeks. The enhancement is attributed to flavonoids, which stimulate neuronal regeneration and improve cellular communications.
A University of Missouri researcher found a way to isolate and test genes related to memory by manipulating compounds associated with the fly brain. This discovery could lead to new treatments for Parkinson's disease and depression.
Researchers found that acute stress activates corticotropin-releasing hormones, disrupting memory formation by disintegrating dendritic spines. Blocking CRH molecules' interaction can eliminate stress damage to these structures.