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Instituto de Medicina Molecular


New study suggests combination therapy to prevent resistance to treatment in hormone responsive metastatic breast cancer patients

A new study led by Sandra Casimiro and Luís Costa found a promising molecular target, the RANK signaling pathway, that could be used in combination with standard care to avoid resistance to treatment in metastatic breast cancer. High levels of RANK in cancer cells are associated with resistance to treatment with CDK4/6 inhibitors.

SourceInstituto de Medicina Molecular·JournalCell Reports Medicine·TypeExperimental study·DateJul 13, 2023

Getting around muscle aging

A new study found a protein that regulates macrophage function, clearing residues from regenerating muscle and recovering regenerative capacity in aged mice. The discovery holds promise for regenerative medicine and aging, potentially improving the success of current stem-cell based therapies.

SourceInstituto de Medicina Molecular·JournalNature Aging·TypeExperimental study·DateMar 23, 2023

Brain metastasis looking back in the mirror

Researchers created patient-derived models of brain metastases that recapitulate human disseminated disease, reflecting clinical manifestations and biological characteristics. These models can be used to test therapeutic value of different treatments and explore new approaches tailored to each patient.

SourceInstituto de Medicina Molecular·JournalCell Reports Medicine·TypeExperimental study·DateMay 3, 2022

Dressing up RNA molecules to last

A new mechanism has been discovered that decorates the end tails of RNA molecules in a parasite causing sleeping sickness, preventing their degradation and potentially increasing virulence. This fundamental discovery opens new avenues for treatment strategies for this disease, as well as other RNA-based infections/diseases.

SourceInstituto de Medicina Molecular·JournalNature·TypeExperimental study·DateMar 30, 2022

Nuclei on the move for muscle self-repair

A recent study reveals that muscle regeneration after physiological damage relies on the rearrangement of nuclei, accelerating repair of contractile units. Nuclei are attracted to injury sites and deliver mRNA molecules to initiate protein synthesis for muscle repair, offering a new insight into muscle biology.

SourceInstituto de Medicina Molecular·JournalScience·TypeExperimental study·DateOct 14, 2021

Boosting the discovery of new drugs to treat spinal cord injuries using zebrafish

Researchers have designed a simple and efficient platform to discover new drugs treating spinal cord lesions using zebrafish, accelerating the translation period from discovery to clinics. The platform has identified a molecule with motor recovery properties in zebrafish larvae and showed efficacy in spinal cord injury models in rodents.

SourceInstituto de Medicina Molecular·JournalScientific Reports·DateJul 19, 2019

It takes a village...

Researchers identify myofibroblasts as key players in activating nucleus movement, potentially leading to new therapeutic strategies for muscle disorders. The discovery could improve understanding of muscle differentiation, functionality, and regeneration.

SourceInstituto de Medicina Molecular·JournalDevelopmental Cell·DateJul 9, 2018

When low batteries are a good thing

Researchers discovered that the mitochondria of gut-resident white blood cells have a different composition that reduces their energy production, keeping them in a controlled activated state. This knowledge can lead to new diagnostic markers and treatments for conditions like gut inflammations and infections.

SourceInstituto de Medicina Molecular·JournalScience Immunology·DateJun 22, 2018

New mutations in leukemia: Researchers found mechanism that can help design future therapies

A group of mutations in the interleukin-7 receptor gene have been identified in T-cell acute lymphoblastic leukemia patients, leading to uncontrolled cell proliferation. Researchers found that certain pharmaceutical drugs already in clinical use can eliminate these cells, providing a potential therapeutic approach against leukemia.

SourceInstituto de Medicina Molecular·JournalNature Genetics·DateSep 4, 2011