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Temple University Health System


Research maps key signaling pathways linking calcium entry and exit in activated T cells

A study by Temple-led researchers describes a unique mechanism for coordinating calcium entrance and exit 'doors' on T cells, which helps them carry out their jobs and ensure normal immune function. The research offers new insight into calcium signaling that could help scientists better understand autoimmune and immunodeficiency diseases.

SourceTemple University Health System·JournalScience Signaling·DateOct 8, 2019

New clinical trial analyses predict response to benralizumab by patients with COPD

A new analysis of two international clinical trials predicts that benralizumab may be effective for a subpopulation of patients with moderate to very severe COPD who have elevated blood eosinophil counts and a history of frequent exacerbations. The study found no significant efficacy in reducing annual COPD exacerbation rates for patie...

SourceTemple University Health System·JournalThe Lancet Respiratory Medicine·DateSep 30, 2019

Studies: Benralizumab not effective reducing exacerbations in moderate to very severe COPD

A recent study published in the New England Journal of Medicine found that benralizumab, an asthma drug, was ineffective in reducing exacerbations in patients with moderate to very severe COPD. The study involved over 3,000 patients and showed no significant decrease in COPD exacerbation rates among those receiving benralizumab.

SourceTemple University Health System·JournalNew England Journal of Medicine·DateMay 20, 2019

Novel target identified for neuron regeneration, functional recovery in spinal cord injury

Scientists at Temple University Health System have identified LKB1 as a critical regulator of axon regeneration in mature neurons, leading to significant gains in functional recovery in mice with spinal cord injuries. Targeted upregulation of LKB1 protein stimulated long-distance neuron regeneration and improved locomotor function.

SourceTemple University Health System·JournalMolecular Therapy·DateNov 19, 2018

Elevated homocysteine identified as metabolic risk factor for neurodegenerative diseases

Researchers found that high homocysteine levels in mice lead to increased tau tangles, nerve cell death, and impaired learning and memory. The study suggests a potential link between hyperhomocysteinemia and neurodegenerative diseases, highlighting the need for further investigation into the role of 5-lipoxygenase in disease progression.

SourceTemple University Health System·JournalMolecular Psychiatry·DateMay 16, 2018